提出可同时收缩与伸展的人工拮抗肌,实现无松弛的完整运动范围。
Stretchable Electrohydraulic Artificial Muscle for Full Motion Ranges in Musculoskeletal Antagonistic Joints
- 用非伸缩式电液压驱动器与静电离合器构建拮抗系统。
- 在3.2赫兹频率下实现平滑无缝的双向运动切换。
- 适用于多种人工肌肉,推动假肢与仿生机器人发展。
人工肌肉在模拟生物肌肉力输出功能方面对骨骼肌机器人和假肢至关重要。然而,现有系统通常仅支持收缩或伸展,无法同时实现两者,限制了全功能人工骨骼肌系统的开发。本文提出一种兼具收缩与伸展能力的人工拮抗肌系统。设计将非伸缩式电液压软执行器(HASELs)与静电离合器集成于拮抗骨骼肌框架中,使关节在不因肌腱松弛导致位移损失的情况下实现完整运动范围。通过同步方法协调肌体与离合器单元,确保平滑运动轨迹与速度,支持最高3.2赫兹的操作频率。尽管原型使用电液压执行器,该肌肉-离合器概念亦可适配其他非伸缩型人工肌肉,如McKibben执行器,拓展其在拮抗结构中的伸展能力与全运动范围性能。本设计显著推进了更高效、更类生物的人工骨骼肌系统核心组件的发展。
原文摘要 · Abstract (English)
Artificial muscles play a crucial role in musculoskeletal robotics and prosthetics to approximate the force-generating functionality of biological muscle. However, current artificial muscle systems are typically limited to either contraction or extension, not both. This limitation hinders the development of fully functional artificial musculoskeletal systems. We address this challenge by introducing an artificial antagonistic muscle system capable of both contraction and extension. Our design integrates non-stretchable electrohydraulic soft actuators (HASELs) with electrostatic clutches within an antagonistic musculoskeletal framework. This configuration enables an antagonistic joint to achieve a full range of motion without displacement loss due to tendon slack. We implement a synchronization method to coordinate muscle and clutch units, ensuring smooth motion profiles and speeds. This approach facilitates seamless transitions between antagonistic muscles at operational frequencies of up to 3.2 Hz. While our prototype utilizes electrohydraulic actuators, this muscle-clutch concept is adaptable to other non-stretchable artificial muscles, such as McKibben actuators, expanding their capability for extension and full range of motion in antagonistic setups. Our design represents a significant advancement in the development of fundamental components for more functional and efficient artificial musculoskeletal systems, bringing their capabilities closer to those of their biological counterparts.
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